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The accepted and most common treatment for Lyme Disease is antibiotics. Patients usually receive multiple rounds of various antibiotics in order to directly target the bacterial spirochete. Some patients respond positively to this type of therapy while others do not.
This direct approach, although beneficial for some, may be deleterious for others, and does not address many potential underlying issues that contribute to Lyme Disease and prevent people from successfully overcoming their disabling symptoms and resuming a normal, productive life.
The most common early symptom post-tick bite is the bulls-eye or target rash, also known as Erythema Migrans. This is often accompanied with flu-like symptoms such as fever, fatigue and general achiness. However, many people diagnosed with Lyme Disease do not recall experiencing a tick bite and did not experience a bulls-eye rash, and therefore go undiagnosed and untreated, often for years.
At Anatara Medicine, we use advanced laboratory testing such as IGeneX, iLymeSpot and Fry Labs to diagnosis Lyme disease along with additional lab markers to assess immune function and inflammation, such as hsCRP, CD57, C4a, C3a, etc.
PEOPLE ARE NOT JUST THEIR LABORATORY RESULTS.
Along with laboratory markers we also use symptom and quality of life questionnaires to monitor symptom changes to ensure effective therapy over the course of treatment.
Lyme disease is not just an infection and is commonly not successfully treated with target therapies alone. Symptoms of Lyme disease can also be mistaken for a variety of other diseases as it can negatively impact multiple aspects of health.
We routinely assess and optimize the following parameters of health in order to successfully treat Lyme Disease and reduce inflammatory triggers:
Digestive health (Leaky Gut, Candida, SIBO, etc)
Detoxification and cellular repair pathways
Heavy metal toxicity
Genetic testing (immune susceptibility to infections and mold, methylation defects)
Denise D. was treated for Lyme disease with the Hermann device, multi-pass ozone therapy as well as supportive intravenous therapies. She shares her story in this video.
Each person is unique and responds differently to infection and treatment. There is no single treatment that works for everyone. Therefore, each person requires a customized treatment plan to match his or her physiological needs. Visit Our Team to learn more about our outstanding practitioners.
Unlike conventional treatment protocols, Anatara offers both targeted Lyme disease treatments and supportive treatments to optimize other systems of the body. Although, we do not use antibiotics in the treatment of Lyme disease we work with patients who are currently on them or have previously been on them.
Core treatments of our program are listed below. These are used in conjunction with other therapies to address adrenal, thyroid and digestive health, detoxification, inflammation, nutrient deficiencies and more.
IV ozone (major autohemotherapy), multipass or hyperbaric IV ozone therapy, ozone insufflation, and ozone sauna.
Ultraviolet light therapy is an uncomplicated, painless, and safe means of treating disease.
We will prescribe you an individualized herbal formula.
IV Silver, high dose Vitamin C, Alpha Lipoic Acid, phosphatidyl choline, various multi-mineral and multi-vitamin IVs, customized IVs, etc.
Complement Split Products C3a and C4a in Chronic Lyme Disease
Complement split products C3a and C4a are reportedly elevated in patients with acute Lyme disease. We have now examined these immunologic markers in patients with chronic Lyme disease compared to appropriate disease controls. The study population consisted of 29 healthy controls, 445 patients with chronic Lyme disease, 11 patients with systemic lupus erythematosus (SLE) and six patients with AIDS.
The Lyme disease patients were divided according to predominant musculoskeletal symptoms (324 patients) or predominant neurologic symptoms (121 patients). C3a and C4a levels were measured by radioimmunoassay. All patients with chronic Lyme disease and AIDS had normal C3a levels compared to controls, whereas patients with SLE had significantly increased levels of this marker.